Friday, March 8, 2013

Dogs may have been used to make Petfood and animal feed


Dogs may have been used to make Petfood and animal feed

Process Management feed contamination dogs statistics petfood markets and trade processing sheep Spain 6030

A major police investigation is underway in Spain since last year into allegations that stray and abandoned dogs have ended up in petfood and livestock feed, a Spanish newspaper reported.

According to reports a criminal gang in Spain took the bodies of dogs and other animals from animal sanctuaries, vets, zoos and farms, awaiting incineration, and processed them to create protein and fats that could be sold on.

Last year, police found a warehouse filled with 15 tonnes of dead stray dogs which they believe were going to be processed into animal feed, in the Galician town of As Neves. Similar stocks have also been found in other warehouses in Northern Spain.

Seprona, the environmental arm of the Guardia Civil, has sent samples of commercial pet food to the Anfaco-Cecopesca laboratories in Vigo, Galicia, after a judge received reports from an industry whistle-blower.

Laboratory tests of fat samples, intended for animal feed, at one of the processing plants based in the town of Aldeaseca de la Frontera, in Salamanca, there were DNA traces of both sheep and dog.

Scottish Labour MEP, Alyn Smith, said: ‘These revelations from Spain indicate just where I fear this may be going. By the time meat becomes "protein" then traceability all but breaks down, especially in the pet and animal feed markets.

‘I'm concerned that given the EU-wide pet food market this contamination could be considerably more widespread.

‘The spectre of forced cannibalism turns this issue into something considerably more serious, and we need reassurance that this is an isolated incident of criminality, albeit it would seem on a pretty significant scale given the reported sourcing of 15 tonnes of dogs must take some organisation.’

Source: DailyMail

by AllAboutFeed 8 Mar 2013


DEFRA Department for Environment, Food & Rural Affairs

Area 307, London, SW1P 4PQ Telephone: 0207 904 6000 Direct line: 0207 904 6287 E-mail:


Mr T S Singeltary P.O. Box 42 Bacliff Texas USA 77518

21 November 2001

Dear Mr Singeltary


Thank you for e-mail regarding the hounds survey. I am sorry for the long delay in responding.

As you note, the hound survey remains unpublished. However the Spongiform Encephalopathy Advisory Committee (SEAC), the UK Government's independent Advisory Committee on all aspects related to BSE-like disease, gave the hound study detailed consideration at their meeting in January 1994. As a summary of this meeting published in the BSE inquiry noted, the Committee were clearly concerned about the work that had been carried out, concluding that there had clearly been problems with it, particularly the control on the histology, and that it was more or less inconclusive. However was agreed that there should be a re-evaluation of the pathological material in the study.

Later, at their meeting in June 95, The Committee re-evaluated the hound study to see if any useful results could be gained from it. The Chairman concluded that there were varying opinions within the Committee on further work. It did not suggest any further transmission studies and thought that the lack of clinical data was a major weakness.

Overall, it is clear that SEAC had major concerns about the survey as conducted. As a result it is likely that the authors felt that it would not stand up to r~eer review and hence it was never published. As noted above, and in the detailed minutes of the SEAC meeting in June 95, SEAC considered whether additional work should be performed to examine dogs for evidence of TSE infection. Although the Committee had mixed views about the merits of conducting further work, the Chairman noted that when the Southwood Committee made their recommendation to complete an assessment of possible spongiform disease in dogs, no TSEs had been identified in other species and hence dogs were perceived as a high risk population and worthy of study. However subsequent to the original recommendation, made in 1990, a number of other species had been identified with TSE ( e.g. cats) so a study in hounds was less

As this study remains unpublished, my understanding is that the ownership of the data essentially remains with the original researchers. Thus unfortunately, I am unable to help with your request to supply information on the hound survey directly. My only suggestion is that you contact one of the researchers originally involved in the project, such as Gerald Wells. He can be contacted at the following address.

Dr Gerald Wells, Veterinary Laboratories Agency, New Haw, Addlestone, Surrey, KT 15 3NB, UK

You may also wish to be aware that since November 1994 all suspected cases of spongiform encephalopathy in animals and poultry were made notifiable. Hence since that date there has been a requirement for vets to report any suspect SE in dogs for further investigation. To date there has never been positive identification of a TSE in a dog.

I hope this is helpful

Yours sincerely 4




I am sorry, but I really could have been a co-signatory of Gerald's minute.

I do NOT think that we can justify devoting any resources to this study, especially as larger and more important projects such as the pathogenesis study will be quite demanding.

If there is a POLITICAL need to continue with the examination of hound brains then it should be passed entirely to the VI Service.

J W WILESMITH Epidemiology Unit 18 October 1991

Mr. R Bradley

cc: Mr. G A H Wells

3.3. Mr R J Higgins in conjunction with Mr G A Wells and Mr A C Scott would by the end of the year, indentify the three brains that were from the ''POSITIVE'' end of the lesion spectrum.

TSE in dogs have not been documented simply because OF THE ONLY STUDY, those brain tissue samples were screwed up too. see my investigation of this here, and to follow, later follow up, a letter from defra, AND SEE SUSPICIOUS BRAIN TISSUE SAF's. ...TSS


GAH WELLS (very important statement here...TSS)


AS implied in the Inset 25 we must not _ASSUME_ that transmission of BSE to other species will invariably present pathology typical of a scrapie-like disease.


76 pages on hound study;


The spongiform changes were not pathognomonic (ie. conclusive proof) for prion disease, as they were atypical, being largely present in white matter rather than grey matter in the brain and spinal cord. However, Tony Scott, then head of electron microscopy work on TSEs, had no doubt that these SAFs were genuine and that these hounds therefore must have had a scrapie-like disease. I reviewed all the sections myself (original notes appended) and although the pathology was not typical, I could not exclude the possibility that this was a scrapie-like disorder, as white matter vacuolation is seen in TSEs and Wallerian degeneration was also present in the white matter of the hounds, another feature of scrapie.

38.I reviewed the literature on hound neuropathology, and discovered that micrographs and descriptive neuropathology from papers on 'hound ataxia' mirrored those in material from Robert Higgins' hound survey. Dr Tony Palmer (Cambridge) had done much of this work, and I obtained original sections from hound ataxia cases from him. This enabled me provisionally to conclude that Robert Higgins had in all probability detected hound ataxia, but also that hound ataxia itself was possibly a TSE. Gerald Wells confirmed in 'blind' examination of single restricted microscopic fields that there was no distinction between the white matter vacuolation present in BSE and scrapie cases, and that occurring in hound ataxia and the hound survey cases.

39.Hound ataxia had reportedly been occurring since the 1930's, and a known risk factor for its development was the feeding to hounds of downer cows, and particularly bovine offal. Circumstantial evidence suggests that bovine offal may also be causal in FSE, and TME in mink. Despite the inconclusive nature of the neuropathology, it was clearly evident that this putative canine spongiform encephalopathy merited further investigation.

40.The inconclusive results in hounds were never confirmed, nor was the link with hound ataxia pursued. I telephoned Robert Higgins six years after he first sent the slides to CVL. I was informed that despite his submitting a yearly report to the CVO including the suggestion that the hound work be continued, no further work had been done since 1991. This was surprising, to say the very least.

41.The hound work could have provided valuable evidence that a scrapie-like agent may have been present in cattle offal long before the BSE epidemic was recognised. The MAFF hound survey remains unpublished.

Histopathological support to various other published MAFF experiments

42.These included neuropathological examination of material from experiments studying the attempted transmission of BSE to chickens and pigs (CVL 1991) and to mice (RVC 1994).

It was thought likely that at least some, and probably all, of the cases in zoo animals were caused by the BSE agent. Strong support for this hypothesis came from the findings of Bruce and others (1994) ( Bruce, M.E., Chree, A., McConnell, I., Foster, J., Pearson, G. & Fraser, H. (1994) Transmission of bovine spongiform encephalopathy and scrapie to mice: strain variation and species barrier. Philosophical Transactions of the Royal Society B 343, 405-411: J/PTRSL/343/405 ), who demonstrated that the pattern of variation in incubation period and lesion profile in six strains of mice inoculated with brain homogenates from an affected kudu and the nyala, was similar to that seen when this panel of mouse strains was inoculated with brain from cattle with BSE. The affected zoo bovids were all from herds that were exposed to feeds that were likely to have contained contaminated ruminant-derived protein and the zoo felids had been exposed, if only occasionally in some cases, to tissues from cattle unfit for human consumption.




worse still, there is serious risk the media could get to hear of such a meeting...


Crushed heads (which inevitably involve brain and spinal cord material) are used to a limited extent but will also form one of the constituent raw materials of meat and bone meal, which is used extensively in pet food manufacturer...

2. The Parliamentary Secretary said that he was concerned about the possibility that countries in which BSE had not yet been detected could be exporting raw meat materials (in particular crushed heads) contaminated with the disease to the UK for use in petfood manufacture...


YOU explained that imported crushed heads were extensively used in the petfood industry...

In particular I do not believe one can say that the levels of the scrapie agent in pet food are so low that domestic animals are not exposed...

some 100+ _documented_ TSE cats of all types later...tss

on occassions, materials obtained from slaughterhouses will be derived from sheep affected with scrapie or cattle that may be incubating BSE for use in petfood manufacture...

*** Meldrum's notes on pet foods and materials used

*** BSE & Pedigree Petfoods ***


July 20, 200 1 Date: Thu, 16 Aug 2001 13:52:58 –0400

From: "Cook, Nancy" Reply-To: Bovine Spongiform Encephalopathy


######## Bovine Spongiform Encephalopathy #########

Robert, just wanted to comment on your request that the "Do not feed to Cattle or other Ruminants" statement be placed on all animal feeds. In 1997, we undertook a broad, five city survey to determine what effect that statement might have in the marketplace if it occurred on pet food labels.

Overwhelmingly, and in all locations, an immediate and severe effect was projected, not only into pet food, but into the Meat Counter as well, as people struggled with the idea that "if it's not good for ruminants (whatever they are?), why should I feed it to my pets, and oh, by the way, why should I eat beef at all if it's a problem?"

The Office of Management and Budget agreed with our findings and advised FDA that the labeling was not needed on pet food for retail sale or for laboratory animal feed. However, salvage products are required to bear the statement, since those products are often used for swine feed.

In most states, pets are classified as dogs and cats. Specialty pets are other caged and "aquariumed" critters. Horses and rabbits are classified as livestock.

Hope this is helpful.

Nancy K. Cook Pet Food Institute 2025 M Street, Suite 800 Washington, DC 20036 202-367-1120 202-367-2120 (fax)


Date: Fri, 17 Aug 2001 14:37:50 –0700

From: "Terry S. Singeltary Sr."

Reply-To: Bovine Spongiform Encephalopathy

To: References: 1

######## Bovine Spongiform Encephalopathy #########

Greetings again List Members,

here is a bit of what was thought of pet foods and TSEs in the early days of the BSE Inquiry;

What is meat and other material from scrapie-infected sheep used for - does it include pet food and material for biological products?

Pet Food

As initial preclinical multiplication of the agent takes place in the spleen and other parts of the lympho-reticular system (LRS) there is obviously the possibility that scrapie infected material is used for pet food in addition to material from clinically affected sheep. Sheep spleens are used exclusively for pet foods and processed sheep heads are undoubtedly included.

Commercial canned pet food is subject to heat treatment. The following treatments are employed by . . .

[A table has been deleted here for commercial-in-confidence reasons.]



Saturday, August 29, 2009

FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009

Thursday, September 3, 2009

429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

Tuesday, November 3, 2009

re-FOIA REQUEST ON FEED RECALL PRODUCT contaminated with prohibited material Recall # V-258-2009 and Recall # V-256-2009

From: Terry S. Singeltary Sr.

Cc: ; ; ;

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

September 4, 2009


Food and Drug Administration

Division of Freedom of Information (HFI-35)

Office of Shared Services

Office of Public Information and Library Services

5600 Fishers Lane

Rockville, MD 20857

Or requests may be sent via fax to: fax number 301-443-1726 or 301-443-1719. If experience difficulty sending a fax, please call (301) 443-2414.


Terry S. Singeltary Sr.

P.O. Box 42

Bacliff, Texas USA 77518

Greetings FDA FOIE, and the Honorable Phyllis Fong et al @ OIG FOIA,



Canine Spongiform Encephalopathy CSE TSE

>>> Is anybody even looking at the dogs..especially with CWD now so widespread? <<<

NA, na, na........they know what they will find, Canine Spongiform Encephalopathy, and it was documented, but then they decided not to push the issue anymore, they had enough mad cow disease in different species to deal with. so they screwed the brains up with dogs and deer in the UK. then we took a page or two from the UKs testing protocols and USDA screwed the brains up with cattle, again, and again, and again. then played the stupid card. ya can't fix stupid. ... TSS

Monday, March 8, 2010

Canine Spongiform Encephalopathy aka MAD DOG DISEASE


Another Big Myth about Transmissible Spongiform Encephalopathy, is that TSE will not transmit to dogs. This is simply NOT TRUE. IT is perfectly legal to feed dogs and cats here in the USA bovine meat and bone meal. Canine dementia is real. how many dogs and cats here in the USA are tested for mad cow disease ? I just received this F.O.I.A. request, and thought I would post it here with a follow up on MAD DOG DISEASE. This is a follow up with additional data I just received on a FOIA request in 2009 ;

see full text, and be sure to read the BSE Inquiry documents toward the bottom ;

Monday, March 8, 2010

UPDATE 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

Monday, March 1, 2010


snip...see more here;

Thursday, January 5, 2012

Horse Meat, slaughter for consumption USA

Saturday, August 4, 2012

*** Final Feed Investigation Summary - California BSE Case - July 2012

It was proven in Oprah Winfrey's trial, that Cactus Cattle feeders, sent neurologically ill cattle, some with encephalopathy stamped on the dead slips, were picked up and sent to the renders, along with sheep carcasses.

Web posted Friday, January 23, 1998 5:49 a.m. CT

Witness testifies some ill cattle sent to rendering plant


Globe-News Staff Writer

Witness testifies some ill cattle sent to rendering plant

By CHIP CHANDLER Globe-News Staff Writer


Mike Engler -- son of Paul Engler, the original plaintiff and owner of Cactus Feeders Inc. -- agreed that more than 10 cows with some sort of central nervous system disorder were sent to Hereford By-Products.

The younger Engler, who has a doctorate in biochemistry from Johns Hopkins University, was the only witness jurors heard Thursday in the Oprah Winfrey defamation trial. His testimony will resume this morning.

According to a U.S. Department of Agriculture report from which Winfrey attorney Charles Babcock quoted, encephalitis caused by unknown reasons could be a warning sign for bovine spongiform encephalopathy, or mad cow disease.

Encephalitis was indicated on the death certificates -- or ``dead slips'' -- of three Cactus Feeders cows discussed in court. The slips then were stamped, ``Picked up by your local used cattle dealer'' before the carcasses were taken to the rendering plant.


Web posted Wednesday, February 18, 1998 2:02 p.m. CT

Graphic pictures greet Winfrey jury

By KAY LEDBETTER Globe-News Farm and Ranch Editor

Pictures of sheep heads, euthanized pets and roadkill greeted jurors this morning as they returned to the continuation of the cattlemen vs. Oprah Winfrey lawsuit.

The lawsuit continues today in U.S. District Mary Lou Robinson's court, but in a much diminished state.


Defense lawyer Charles Babcock called Van Smith, a City Paper reporter from Baltimore who had written an article on rendering plants in September 1995.

Smith and Babcock went through more than 50 pictures taken as the reporter toured the Valley Proteins plant in Baltimore and followed a rendering truck to the local animal shelter, a sausage plant and a slaughterhouse.

The pictures showed offal being emptied from the slaughterhouses. They showed animal shelter workers in the euthanasia room; barrels of dead animals in a refrigerated room at the animal shelter; waste meat from the sausage plant; and dead sheep from the slaughterhouse.

Web posted Thursday, February 19, 1998 5:32 a.m. CT

Defense opens case

Cattlemen vs. Oprah Winfrey


Globe-News Staff Writer


Van Smith, a reporter with City Paper in Baltimore, testified about an article he wrote on rendering plants. Smith said he saw sheep taken to a plant despite a voluntary ban on using processed sheep in protein-enhanced feed, backing up a statement Lyman made on Winfrey's show.

Under cross-examination, Smith said he was not sure whether the sheep were used for feed or other animal-derived products.


Van Smith, a reporter with City Paper in Baltimore, testified about an article he wrote on rendering plants. Smith said he saw sheep taken to a plant despite a voluntary ban on using processed sheep in protein-enhanced feed, backing up a statement Lyman made on Winfrey's show.

Under cross-examination, Smith said he was not sure whether the sheep were used for feed or other animal-derived products.

FOR IMMEDIATE RELEASE Statement May 4, 2004 Media Inquiries: 301-827-6242 Consumer Inquiries: 888-INFO-FDA

Statement on Texas Cow With Central Nervous System Symptoms On Friday, April 30 th , the Food and Drug Administration learned that a cow with central nervous system symptoms had been killed and shipped to a processor for rendering into animal protein for use in animal feed.

FDA, which is responsible for the safety of animal feed, immediately began an investigation. On Friday and throughout the weekend, FDA investigators inspected the slaughterhouse, the rendering facility, the farm where the animal came from, and the processor that initially received the cow from the slaughterhouse.

FDA's investigation showed that the animal in question had already been rendered into "meat and bone meal" (a type of protein animal feed). Over the weekend FDA was able to track down all the implicated material. That material is being held by the firm, which is cooperating fully with FDA.

Cattle with central nervous system symptoms are of particular interest because cattle with bovine spongiform encephalopathy or BSE, also known as "mad cow disease," can exhibit such symptoms. In this case, there is no way now to test for BSE. But even if the cow had BSE, FDA's animal feed rule would prohibit the feeding of its rendered protein to other ruminant animals (e.g., cows, goats, sheep, bison).

FDA is sending a letter to the firm summarizing its findings and informing the firm that FDA will not object to use of this material in swine feed only. If it is not used in swine feed, this material will be destroyed. Pigs have been shown not to be susceptible to BSE. If the firm agrees to use the material for swine feed only, FDA will track the material all the way through the supply chain from the processor to the farm to ensure that the feed is properly monitored and used only as feed for pigs.

To protect the U.S. against BSE, FDA works to keep certain mammalian protein out of animal feed for cattle and other ruminant animals. FDA established its animal feed rule in 1997 after the BSE epidemic in the U.K. showed that the disease spreads by feeding infected ruminant protein to cattle.

Under the current regulation, the material from this Texas cow is not allowed in feed for cattle or other ruminant animals. FDA's action specifying that the material go only into swine feed means also that it will not be fed to poultry.

FDA is committed to protecting the U.S. from BSE and collaborates closely with the U.S. Department of Agriculture on all BSE issues. The animal feed rule provides crucial protection against the spread of BSE, but it is only one of several such firewalls. FDA will soon be improving the animal feed rule, to make this strong system even stronger.


Monday, March 26, 2012



Friday, November 09, 2012

*** Chronic Wasting Disease CWD in cervidae and transmission to other species

Sunday, November 11, 2012

*** Susceptibilities of Nonhuman Primates to Chronic Wasting Disease November 2012

Friday, December 14, 2012

Susceptibility Chronic Wasting Disease (CWD) in wild cervids to Humans 2005 - December 14, 2012

Saturday, December 15, 2012

Bovine spongiform encephalopathy: the effect of oral exposure dose on attack rate and incubation period in cattle -- an update 5 December 2012

Thursday, February 14, 2013

The Many Faces of Mad Cow Disease Bovine Spongiform Encephalopathy BSE and TSE prion disease

why do we not want to do TSE transmission studies on chimpanzees $

5. A positive result from a chimpanzee challenged severly would likely create alarm in some circles even if the result could not be interpreted for man. I have a view that all these agents could be transmitted provided a large enough dose by appropriate routes was given and the animals kept long enough. Until the mechanisms of the species barrier are more clearly understood it might be best to retain that hypothesis.



Wednesday, February 16, 2011




Sunday, December 12, 2010

EFSA reviews BSE/TSE infectivity in small ruminant tissues News Story 2 December 2010

Sunday, April 18, 2010


Thursday, December 23, 2010

Molecular Typing of Protease-Resistant Prion Protein in Transmissible Spongiform Encephalopathies of Small Ruminants, France, 2002-2009

Volume 17, Number 1 January 2011

Thursday, November 18, 2010

Increased susceptibility of human-PrP transgenic mice to bovine spongiform encephalopathy following passage in sheep

Monday, April 25, 2011

Experimental Oral Transmission of Atypical Scrapie to Sheep

Volume 17, Number 5-May 2011

Friday, February 11, 2011

Atypical/Nor98 Scrapie Infectivity in Sheep Peripheral Tissues

Thursday, March 29, 2012

atypical Nor-98 Scrapie has spread from coast to coast in the USA 2012

NIAA Annual Conference April 11-14, 2011San Antonio, Texas